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ELISA Kit for Complement Receptor 1, Erythrocyte (CR1)
CC-SEB123Hu
Producto nuevo
48T, 96T, 96T×5, 96T×10, 96T×100
Comprando este producto generará 6 Biopuntos. Su cesta contiene un total 6 Biopuntos puede ser convertido en un Biobonos Descuento 24.00EUR.
Hoja técnica
| Size | 96T |
| Storage Conditions | -20°C |
| Shipping Conditions | Entre 4°C y 8°C |
| Applications | Enzyme-linked immunosorbent assay for Antigen Detection. |
| Detection Range | 0.156-10ng/mL |
| Sensitivity | The minimum detectable dose of this kit is typically less than 0.061ng/mL. |
| Research Area | Signal transduction |
| Organism species | Homo sapiens (Human) |
| Alternative Names | CD35 |
| Item Name | Complement Receptor 1, Erythrocyte |
| Assay Length | 3h |
| Method | Double-antibody Sandwich |
| Sample Type | serum, plasma, tissue homogenates, cell lysates, cell culture supernates and other biological fluids |
| Formato | 48T, <i>96T</i>, 96T×5, 96T×10, 96T×100 |
| Test Principle | The test principle applied in this kit is Sandwich enzyme immunoassay. The microtiter plate provided in this kit has been pre-coated with an antibody specific to Complement Receptor 1, Erythrocyte (CR1). Standards or samples are then added to the appropri |
| Assay procedure summary | 1. Prepare all reagents, samples and standards |
| UniProt ID | P17927 |
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CITATIONS
Ghrelin Alleviates Spinal Cord Injury in Rats Via Its Anti-inflammatory Effects;Complement Receptor 1 Gene Variants Are Associated with Erythrocyte Sedimentation Rate;Cerebrospinal fluid levels of complement proteins C3, C4 and CR1 in Alzheimer's disease;High complement levels in astrocyte‐derived exosomes of Alzheimer disease;Human complement receptor type 1 (CR1) protein levels and genetic variants in chronic Chagas Disease;Complement protein levels in plasma astrocyte-derived exosomes are abnormal in conversion from mild cognitive impairment to Alzheimer's disease …;Traumatic brain injury increases plasma astrocyte‐derived exosome levels of neurotoxic complement proteins;Hepatitis B Virus Infection Among Leprosy Patients: A Case for Polymorphisms Compromising Activation of the Lectin Pathway and Complement Receptors;